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Eur J Cardiothorac Surg 2005;28:443-447
© 2005 Elsevier Science NL

Original articles

Mitral valve repair for commissural prolapse: surgical techniques and long term results

^a Department of Cardiovascular Surgery, Pitié Salpêtrière Hospital, Paris, France
^b Department of Biostatistics-Health, UMR CNRS 5558, Lyon, France

Received 1 February 2005; received in revised form 6 May 2005; accepted 9 May 2005.

^* Corresponding author. Address: Department of Cardiovascular Surgery, Hôpital de la Pitié-Salpêtrière, 50-52 boulevard Vincent Auriol, 75013 Paris, France. Tel.: +33 1 42 16 56 85; fax: +33 1 42 16 56 78. (Email: c.acar{at}psl.aphp.fr).

Abstract

Objective: The aim of this study was to describe the pattern of lesions responsible for commissural prolapse, the techniques of valve repair and their long-term results. Methods: Between 1992 and 2004, 128 mitral valve repairs were consecutively performed for commissural prolapse. There were 86 males and 42 females, the median age was 57.5 years (range 14–84 years). Forty-six percent of patients were in NYHA III or IV, mean ejection fraction was 61±9.4%. The diagnosis of commissural prolapse was recognized by preoperative echocardiography in 32% of the patients and was revealed by intraoperative inspection of the valve in the other cases. The site of the prolapse was the posteriomedial commissure (n=94), the anterior commissure (n=30) or both (n=4). The aetiologies were: infective endocarditis (n=56), degenerative (n=46), ischemic (n=25), congenital mitral regurgitation (n=1). The commissural prolapse was associated with another mitral valvular lesion requiring a specific treatment in 61 cases (47.7%). An associated procedure was carried out in 45 patients. Results: The operative treatment of the commissural prolapse included: commissural closure 65 (50.8%), leaflet resection 31 (24.2%), transposition or shortening of chordae 19 (14.8%), reimplantation or shortening of papillary muscles 3 (2.3%), and replacement of the commissural area by a partial mitral homograft 10 (8%). In-hospital mortality included three deaths (2.3%) and four patients (3.1%) were reoperated: three pericardial drainages for hemopericardium and one for mediastinitis. During the follow-up, one patient died (0.8%) from myocardial infarction and eight patients (6.3%) were reoperated including six (4.7%) for recurrent mitral regurgitation. After a median follow-up time of 76.9 months (range from 15 days to 160 months), 116 patients (90.1%) were in NYHA I. Echocardiographs showed no or minimal insufficiency in 112 patients (87.5%) and mild or moderate insufficiency in 10 patients (7.8%). Conclusions: The diagnosis of commissural prolapse is difficult by preoperative echocardiography. The aetiology of the mitral disease is variable (endocarditis, degenerative or ischemic mitral regurgitation). Using a variety of techniques, commissural prolapse can be repaired with excellent clinical and echographic long-term results.

Key Words: Mitral valve repair • Mitral valve regurgitation • Homograft • Endocarditis

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